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Anti-RELA / NFKB p65 Antibody (aa297-308) LS-C9296

Ordering

Wt. Vol. Conc. Price
100 µg - 0.5 mg/ml $635
Inquire for larger quantities

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Rabbit Polyclonal (IgG) to Human RELA / NFKB p65
Human, Monkey, Mouse, Rat, Bat, Bovine, Dog, Hamster, Horse
IHC, Immunofluorescence, Western blot, ELISA
Unconjugated

Details

Human RELA / NFKB p65
Rabbit
Human, Monkey, Mouse, Rat, Bat, Bovine, Dog, Hamster, Horse (tested or 100% immunogen sequence identity)
IgG Polyclonal
Immunoaffinity purified

Applications

IHC (1:100), Immunofluorescence (1:200), Western blot (1:1000), ELISA (1:100 - 1:1000)

Specificity and Use

RELA / NFKB p65 antibody was raised against a synthetic peptide, RIEEKRKRTYET, corresponding to the NLS of the human NFkB p65 protein conjugated to KLH using maleimide. A residue of cysteine was added to the amino terminal end to facilitate coupling. Percent identity by BLAST analysis: Human, Chimpanzee, Gorilla, Orangutan, Gibbon, Monkey, Marmoset, Woolly monkey, Mouse, Rat, Hamster, Elephant, Panda, Porcine, Bovine, Dog, Bat, Horse (100%); Paramecium (83%).
aa297-308
Recognizes the nuclear localization sequence (NLS) NLS of human p65. The epitope overlaps the NLS of the p65 subunit of the NFkB heterodimer.
Suitable for Immunohistochemistry, Immunofluorescence, Western Blot, ELISA and Supershift assays. Western Blot: 1:1000. Immunohistochemistry: 1:100. Immunofluorescence microscopy: 1:100. Super shift: 0.5-1 ul/assay.

Packaging

PBS, pH 7.2, 0.1% sodium azide, 40% glycerol.
Long term: -20°C; Short term: +4°C. Avoid repeat freeze-thaw cycles.
For research use only.

About RELA / NFKB p65

Q04206 NM_021975 NP_068810.3

RELA Antibody, NF-kappa-B p65delta3 Antibody, NFKB3 Antibody, p65 Antibody, Transcription factor p65 Antibody

NF-kappa-B is a pleiotropic transcription factor present in almost all cell types and is the endpoint of a series of signal transduction events that are initiated by a vast array of stimuli related to many biological processes such as inflammation, immunity, differentiation, cell growth, tumorigenesis and apoptosis.

Requested From: United States
Date Requested: 9/29/2016

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