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Anti-GPBAR1 / TGR5 Antibody (Cytoplasmic Domain) IHC-plus™ LS-A1933

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Wt. Vol. Conc. Price
50 µg 50 µl 1 mg/ml $395
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Rabbit Polyclonal to Human GPBAR1 / TGR5
Human
IHC - Paraffin
Unconjugated

Details

Human GPBAR1 / TGR5
Rabbit
Human (tested or 100% immunogen sequence identity)
Monkey, Horse, Rabbit (at least 90% immunogen sequence identity)
Polyclonal
Immunoaffinity purified

Applications

IHC - Paraffin (18 µg/ml)

Specificity and Use

GPBAR1 / TGR5 antibody was raised against synthetic 16 amino acid peptide from 1st cytoplasmic domain of human TGR5. Percent identity with other species by BLAST analysis: Human, Gorilla, Gibbon, Marmoset, Elephant (100%); Monkey, Rabbit, Horse (94%); Mouse, Bat, Bovine, Hamster (88%); Rat, Dog, Opossum (81%).
LS-E28443 - Liquid - 50 µg - $145.00
Cytoplasmic Domain
Human TGR5. BLAST analysis of the peptide immunogen showed no homology with other human proteins.

Packaging

PBS, less than 0.1% sodium azide.
Aliquot and store undiluted at -20°C or below for up to 1 year. Can be stored undiluted at 4°C for up to 1 month. Avoid freeze thaw cycles.
For research use only.

About GPBAR1 / TGR5

Q8TDU6 AB083601 NP_733800.1

GPBAR1 Antibody, BG37 Antibody, GPR131 Antibody, HGPCR19 Antibody, M-BAR Antibody, GPCR19 Antibody, TGR5 Antibody, GPCR TGR5 Antibody, HBG37 Antibody, Membrane bile acid receptor Antibody

GPBAR1 / TGR5 is a member of the G protein-coupled receptor (GPCR) superfamily. This enzyme functions as a cell surface receptor for bile acids. Treatment of cells expressing this GPCR with bile acids induces the production of intracellular cAMP, activation of a MAP kinase signaling pathway, and internalization of the receptor. The receptor is implicated in the suppression of macrophage functions and regulation of energy homeostasis by bile acids.

Immunohistochemistry

Immunohistochemistry
Anti-TGR5 antibody LS-A1933 IHC of human kidney. Immunohistochemistry of formalin-fixed, paraffin-embedded tissue after heat-induced antigen retrieval.
 

Requested From: United States
Date Requested: 9/28/2016

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