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Mouse DLL4 ELISA Kit (Sandwich ELISA) - LS-F24793


Size Price
1 plate $550
Inquire for larger quantities
LSBio (Direct) LSBio (Direct)

Most Popular DLL4 Elisa Kits

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Rat DLL4 ELISA Kit (Sandwich ELISA) - LS-F8044
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Human DLL4 ELISA Kit (Sandwich ELISA) - LS-F17072
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0.156-10 ng/ml
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Sandwich ELISA (enzyme-linked immunosorbent assay) kit
For the detection and quantification of endogenous levels of natural and/or recombinant Mouse DLL4 proteins.
LS-F24793 is a 96-well enzyme-linked immunosorbent assay (ELISA) for the Quantitative detection of Mouse DLL4 in samples of Cell Culture Supernatants, Plasma and Serum. It is based upon a Sandwich assay principle and can be used to detect levels of DLL4 as low as 10 picograms per milliliter.
All LSBio Custom kits have been predesigned and all necessary components, including antibodies and standards have been identified. Upon receiving a custom kit order, the kit will be assembled and quality control tested before being shipped out. Kit assembly and testing typically takes 4 to 6 weeks. In most cases the final kit is based on the Sandwich assay principle, with a few being Competitive EIA based. Specifications such as Range, Sensitivity, and Precision are defined upon completion. In the event that the custom kit cannot be successfully developed with 6 weeks of the order date, the customer will be notified and the order canceled at no cost.
Cell Culture Supernatants, Plasma, Serum
96-Well Strip Plate
Colorimetric - 450nm
156 - 10000 pg/ml
10 pg/ml
Due to their limited shelf life, LSBio ELISA kits are not typically stocked as finished goods. Upon receipt of an order each kit is assembled and tested to ensure that it meets specifications before shipping. Minor changes may occur to the Range, Sensitivity, and Precision. In the event of a significant change the order would be confirmed with the customer before shipping. ELISA kit lot numbers reflect the date of final assembly and testing for each specific kit rather than a bulk manufactured lot. All kits are tested to confirm that they fall within their defined Inter- and Intra- assay coefficient of variation.
  • Coated 96-well Strip Plate
  • Standard (Lyophilized)
  • Biotinylated Detection Antibody (100x)
  • Avidin-Biotin-HRP Complex (100x)
  • Sample Diluent
  • Biotinylated Detection Antibody Diluent
  • Avidin-Biotin-HRP Complex Diluent
  • Wash Buffer (20x)
  • TMB Substrate
  • Stop Solution
  • Adhesive Plate Sealers
    Delta like ligand 4 is a protein that in humans is encoded by the DLL4 gene. It is mapped to 15q15.1. This gene is a homolog of the Drosophila delta gene. DLL4 is a transmembrane ligand for Notch receptors that shows restricted expression to endothelial cells (ECs), in particular to arteries and capillaries, and is involved in vascular development, and it also appeared to be a major trigger of Notch receptor activities previously implicated in arterial and vascular development. Mouse DLL4 could activate mouse Notch1 and mouse Notch4. DLL4 acts as a negative regulator of tumor angiogenesis, its blockade results in the striking uncoupling of tumor growth from vessel density, presenting a novel therapeutic approach even for tumors resistant to anti-VEGF therapies. In addition to it, this gene also plays an important role in promoting Th17 effector activity during mycobacterial challenge.
Store at 4°C for 6 months or at -20°C for one year. Avoid freeze/thaw cycles.
For research use only.

About DLL4

Q9NR61 NM_019074 NP_061947.1

DLL4 Elisa Kit, Delta-like 4 protein Elisa Kit, Delta like 4 Elisa Kit, Delta-like 4 (Drosophila) Elisa Kit, Delta-like 4 homolog Elisa Kit, Drosophila Delta homolog 4 Elisa Kit, Notch ligand DLL4 Elisa Kit, Delta 4 Elisa Kit, Delta ligand 4 Elisa Kit, Delta-like protein 4 Elisa Kit, Delta4 Elisa Kit, Hdelta2 Elisa Kit, Notch ligand delta-2 Elisa Kit

Involved in the Notch signaling pathway as Notch ligand. Activates NOTCH1 and NOTCH4. Involved in angiogenesis; negatively regulates endothelial cell proliferation and migration and angiogenic sprouting. Essential for retinal progenitor proliferation is required for suppressing rod fates in late retinal progenitors as well as for proper generation of other retinal cell types. During spinal cord neurogenesis, inhibits V2a interneuron fate.

Requested From: United States
Date Requested: 10/25/2016

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