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Anti-TREM2 / TREM-2 Antibody (clone 78.18) LS-C188612


Wt. Vol. Conc. Price
100 µg - 1 mg/ml Unavailable

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Rat Monoclonal [clone 78.18] (IgG1) to Mouse TREM2 / TREM-2
Flow Cytometry (applications tested for the base form of this product only)


Mouse TREM2 / TREM-2
Mouse (tested or 100% immunogen sequence identity)
IgG1 Monoclonal [78.18]
Unconjugated. Also available conjugated with RPE, FITC.
Protein G purified
Unmodified. Also available Low Endotoxin.


  • Flow Cytometry (1:25 - 1:200)
  • (applications tested for the base form of this product only)

Specificity and Use

TREM2 / TREM-2 antibody was raised against mouse TREM-2 Fc fusion protein.
Recognizes Triggering receptor expressed on myeloid cells 2 (TREM-2), an activating receptor of the Ig superfamily. TREM-2 forms a receptor signalling complex with DAP12 to trigger the activation of the immune response in macrophages and dendritic cells. It may be involved in chronic inflammation and may promote the production of constitutive rather than inflammatory chemokines and cytokines. TREM-2 may also have a functional role in osteoclast formation. Stimulation of TREM-2 with clone 78.18 enhances multinucleated osteoclast formation in vitro. Clone 78.18 may also be used to block TREM-2 on pre-osteoclasts to inhibit the formation of multinuclear osteoclasts and to inhibit bone resorption by mature osteoclasts.


PBS, 0.09% sodium azide
+4°C or -20°C, Avoid repeated freezing and thawing.
For research use only.

About TREM2 / TREM-2

Q9NZC2 NM_018965 NP_061838.1

TREM2 Antibody, Trem2b Antibody, TREM-2 Antibody, Trem2a Antibody, Trem2c Antibody

TREM2 / TREM-2 is a membrane protein that forms a receptor signaling complex with the TYRO protein tyrosine kinase binding protein. The encoded protein functions in immune response and may be involved in chronic inflammation by triggering the production of constitutive inflammatory cytokines. Defects in this gene are a cause of polycystic lipomembranous osteodysplasia with sclerosing leukoencephalopathy (PLOSL).

Requested From: 
Date Requested: 4/24/2017

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