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Anti-CSPG4 / NG2 Antibody (N-Terminus) LS-C22115


Wt. Vol. Conc. Price
100 µg - 0.5 mg/ml Unavailable

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100% Guaranteed 100% Guaranteed
Mouse Monoclonal (IgG1,k) to Rat CSPG4 / NG2
Immunofluorescence, Western blot, Immunoprecipitation, ELISA (applications tested for the base form of this product only)


Rat CSPG4 / NG2
Rat (tested or 100% immunogen sequence identity)
IgG1,k Monoclonal
Unconjugated. Also available conjugated with AP, APC, Biotin, FITC, HRP, MaxLight 405, MaxLight 490, MaxLight 550, MaxLight 650, MaxLight 750, PE.
Protein A purified


  • Immunofluorescence (1 - 5 µg/ml)
  • Western blot (1 - 3 µg/ml)
  • Immunoprecipitation
  • ELISA (0.1 - 1 µg/ml)
  • (applications tested for the base form of this product only)

Specificity and Use

CSPG4 / NG2 antibody was raised against purified rat NG2 protein.
This antibody consists of a cocktail of four clones, and will react with the N-terminal domain of NG2, the central collagen binding domain and a membrane proximal epitope. On Western blots, it identifies a band at ~300kD. Reactivity has been confirmed with rat B49 glial cell line.
Suitable for use in Immunoprecipitation, Immunofluorescence, ELISA and Western Blot. Immunoprecipitation: 10 ug/IP reaction. Immunofluorescence: 1-5 ug/ml. ELISA: 0.1-1 ug/ml. Western Blot: 1-3 ug/ml.


PBS, pH 7.4, 0.1% sodium azide.
Long term: -20°C; Short term: +4°C. Avoid repeat freeze-thaw cycles.
For research use only.

About CSPG4 / NG2

Q6UVK1 NM_001897 NP_001888.2

CSPG4 Antibody, MCSP Antibody, MEL-CSPG Antibody, MSK16 Antibody, NG2 Antibody, HMW-MAA Antibody, MCSPG Antibody

Proteoglycan playing a role in cell proliferation and migration which stimulates endothelial cells motility during microvascular morphogenesis. May also inhibit neurite outgrowth and growth cone collapse during axon regeneration. Cell surface receptor for collagen alpha 2(VI) which may confer cells ability to migrate on that substrate. Binds through its extracellular N-terminus growth factors, extracellular matrix proteases modulating their activity.

Requested From: 
Date Requested: 4/26/2017

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