LSBio
ACCN1 / ASIC2 Antibody (C-Terminus)

ACCN1 antibody Western blot of THP-1 cell lysate. This image was taken for the unconjugated form of this product. Other forms have not been tested.

ACCN1 / ASIC2 Antibody (C-Terminus)

Primary AntibodyLSBio Guarantee
Antibody:Rabbit Polyclonal
Applications:Immunohistochemistry (general), Western Blot
Reactivity:Human
Format:Liquid
See all Human ACCN1 / ASIC2 antibodies →
$433.00each
Catalog Number: LS-C110053-100

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Overview

Description
ASIC2 antibody LS-C110053 is an unconjugated rabbit polyclonal antibody to human ASIC2 (ACCN1) (C-Terminus). Validated for WB. Cited in 1 publication.
Application
IHC, WB
Reactivity
Hu
Intended Use
Research Use Only
Guarantees
This antibody carries the LSBio 100% Guarantee.
Category
Antibodies > Primary Antibodies > Polyclonal Antibodies
Alternative Names
ACCN1 | ASIC2a | ASIC2 | Brain sodium channel 1 | Degenerin | ACCN | Acid-sensing ion channel 2 | Mammalian degenerin homolog | MDEG | MDEG1 | MDEG2 | HBNaC1 | BNaC1 | MAMMALIAN DEGENERIN | ASIC2b
UniProt Number
Q16515
SwissProt
Q16515

Target

Target
Human ACCN1 / ASIC2
Antigen Species
Human
Epitope
Human ACCN1
Gene Name
ASIC2

Antibody

Host Species
Rabbit
Clonality
Polyclonal

Immunogen

Immunogen
Synthetic peptide from C-Terminus of human ASIC2 (Q16515, NP_001085). Percent identity by BLAST analysis: Human, Chimpanzee, Gibbon, Monkey, Galago, Marmoset, Mouse, Rat, Elephant, Bovine, Rabbit, Horse, Opossum, Turkey, Chicken (100%); Bat (92%); Zebrafish (85%).
Immunogen Type
Synthetic Peptide (Linear)

Format

Conjugate/Label
Unconjugated
Format
Liquid
Formulation
PBS, 0.09% sodium azide, 2% sucrose
Concentration
0.5 mg/mL
Volume
100 Microliter
Preservative
Yes
Purification Method
Affinity Purified

Storage & Handling

Storage Conditions
Short term: Store at 2-8°C. Long term: Aliquot and store at -20°C. Avoid freeze/thaw cycles.
Recommended Storage Buffer
PBS
Endomorphins potentiate acid-sensing ion channel currents and enhance the lactic acid-mediated increase in arterial blood pressure: effects amplified in hindlimb ischaemia. Mohamed Farrag, Julie K Drobish, Henry L Puhl, Joyce S Kim, Paul B Herold, Marc P Kaufman, Victor Ruiz-Velasco. The Journal of physiology. 2017 December;595:7167-7183. PubMed: 29044528 PMC: PMC5709321

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