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Anti-B1R / BDKRB1 Antibody (Extracellular Domain) IHC-plus™ LS-A3579


Wt. Vol. Conc. Price
50 µg 50 µl 1 mg/ml Unavailable
Same day shipping In Stock - Same day shipping

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Rabbit Polyclonal to Human B1R / BDKRB1
Human, Monkey
IHC - Paraffin


Human B1R / BDKRB1
Human, Monkey (tested or 100% immunogen sequence identity)
Immunoaffinity purified


IHC - Paraffin (20 µg/ml)

Specificity and Use

B1R / BDKRB1 antibody was raised against synthetic 18 amino acid peptide from 2nd extracellular domain of human BDKRB1. Percent identity with other species by BLAST analysis: Human, Gorilla, Monkey (100%); Gibbon (94%); Rat, Hamster (89%); Mouse, Rabbit, Horse (83%).
LS-E28783 - Liquid - 50 µg - $145.00
Immunizing peptide used to generate LS-A3579. Useful for pre-absorption and neutralization of the antibody's antigen binding site.
Extracellular Domain
Human BDKRB1. BLAST analysis of the peptide immunogen showed no homology with other human proteins.


PBS, less than 0.1% sodium azide.
Aliquot and store undiluted at -20°C or below for up to 1 year. Can be stored undiluted at 4°C for up to 1 month. Avoid freeze thaw cycles.
For research use only.

About B1R / BDKRB1

P46663 NM_000710 NP_000701.2

BDKRB1 Antibody, BKB1R Antibody, Bradykinin receptor 1 Antibody, B1BKR Antibody, B1R Antibody, Bk b1 receptor Antibody, BK-1 receptor Antibody, BRADYB1 Antibody, Bradykinin receptor B1 Antibody, Kinin b1 receptor Antibody, B1 bradykinin receptor Antibody, BKR1 Antibody, Bradykinin B1 receptor Antibody

Bradykinin, a 9 aa peptide, is generated in pathophysiologic conditions such as inflammation, trauma, burns, shock, and allergy. Two types of G-protein coupled receptors have been found which bind bradykinin and mediate responses to these pathophysiologic conditions. The protein encoded by this gene is one of these receptors and is synthesized de novo following tissue injury.


Anti-BDKRB1 antibody LS-A3579 IHC of human smooth muscle. Immunohistochemistry of formalin-fixed, paraffin-embedded tissue after heat-induced antigen retrieval.

Requested From: 
Date Requested: 4/27/2017

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