Work with LifeSpan to design a custom immunohistochemistry to address your specific biological question. Outsource the entire localization process without having to
worry about finding and characterizing target specific antibodies, sourcing and validating difficult-to-find tissues, and having the ability to interpret the resulting
immunostaining in relation to complex human pathologies.
Test your therapeutic antibodies in immunohistochemistry against a broad panel of normal frozen human tissue types in order to determine potential unintended binding.
Our non-GLP TCR services are designed on the FDA recommendation outlined in their "Points to Consider in the Manufacture and Testing of Monoclonal Antibody Products for Human Use".
LS-F38847 is a 96-well enzyme-linked immunosorbent assay (ELISA) for the Quantitative detection of Human Fragilis / IFITM3 in samples of Cell Lysates and Tissue Homogenates. It is based upon a Sandwich CLIA assay principle and can be used to detect levels of Fragilis / IFITM3 as low as 11.3 picograms per milliliter.
IFITM3, 1-8U, Dispanin subfamily A member 2b, DSPA2b, Fragilis, IP15
Intended Sample Types
Cell Lysates, Tissue Homogenates
96-Well Strip Plate
27.4 - 20000 pg/ml
Intra-Assay: CV<10% Inter-Assay: CV<12%
Short term: 4°C; Long term: see manual.
Due to their limited shelf life, LSBio ELISA kits are not typically stocked as finished goods. Upon receipt of an order each kit is assembled and tested to ensure that it meets specifications before shipping. Minor changes may occur to the Range, Sensitivity, and Precision. In the event of a significant change the order would be confirmed with the customer before shipping ELISA kit lot numbers reflect the date of final assembly and testing for each specific kit rather than a bulk manufactured lot. All kits are tested to confirm that they fall within their defined Inter- and Intra- assay coefficient of variation.
IFN-induced antiviral protein which disrupts intracellular cholesterol homeostasis. Inhibits the entry of viruses to the host cell cytoplasm by preventing viral fusion with cholesterol depleted endosomes. May inactivate new enveloped viruses which buds out of the infected cell, by letting them go out with a cholesterol depleted membrane.