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Anti-TRA2B / SFRS10 Antibody LS-C10330

Ordering
Wt. Vol. Conc. Price
50 µg - 0.9 mg/ml $335
100 µg - - $455
Inquire for larger quantities

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Related Products

21 TRA2B / SFRS10 Antibodies
2 TRA2B / SFRS10 Proteins
2 TRA2B / SFRS10 Blocking Peptides

100% Guaranteed 100% Guaranteed
Rabbit Polyclonal (IgG) to Human TRA2B / SFRS10
Human, Monkey, Mouse, Rat, Bat, Bovine, Dog, Hamster, Horse, Rabbit, Chicken
IHC, ELISA
Unconjugated

Details

Human TRA2B / SFRS10
Rabbit
Human, Monkey, Mouse, Rat, Bat, Bovine, Dog, Hamster, Horse, Rabbit, Chicken (tested or 100% immunogen sequence identity)
IgG Polyclonal
Immunoaffinity purified

Applications

IHC, ELISA

Specificity and Use

TRA2B / SFRS10 antibody was raised against synthetic peptide: RSYSRDYRRRHSHS (KLH). Percent identity by BLAST analysis: Human, Gorilla, Orangutan, Gibbon, Monkey, Marmoset, Mouse, Rat, Hamster, Elephant, Panda, Dog, Bat, Bovine, Horse, Rabbit, Turkey, Chicken, Platypus, Lizard (100%); Opossum (93%); Xenopus (86%).
Recognizes human Splicing Factor, Arginine/Serine-Rich 10. Species cross-reactivity: rodent.
Suitable for use in ELISA and Immunohistochemistry. ELISA (serum titer): 1:112000. Predicted Molecular Weight: 33.67kD.

Packaging

PBS, pH 7.2, sodium azide, 40% glycerol.
Long term: -20°C; Short term: +4°C. Avoid repeat freeze-thaw cycles.
For research use only.

About TRA2B / SFRS10

P62995 NM_004593 NP_004584.1

TRA2B Antibody, Htra2-beta Antibody, SRFS10 Antibody, TRA2-BETA Antibody, TRAN2B Antibody, SFRS10 Antibody, TRA-2 beta Antibody

Sequence-specific RNA-binding protein which participates in the control of pre-mRNA splicing. Can either activate or suppress exon inclusion. Acts additively with RBMX to promote exon 7 inclusion of the survival motor neuron SMN2. Activates the splicing of MAPT/Tau exon 10. Alters pre-mRNA splicing patterns by antagonizing the effects of splicing regulators, like RBMX. Binds to the AG-rich SE2 domain in the SMN exon 7 RNA. Binds to pre-mRNA.
(More About TRA2B / SFRS10)
Requested From: United States
Date Requested: 10/1/2016
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