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Anti-TNFRSF21 / DR6 Antibody (aa42-56) LS-C14933

Ordering

Wt. Vol. Conc. Price
100 µg - - $445
Inquire for larger quantities

LSBio (Direct) LSBio (Direct)
206-374-1102
866-206-6909
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Most Popular TNFRSF21 / DR6 Antibodies

Anti-TNFRSF21 / DR6 Antibody (aa372-665) LS-C14935
Rabbit Polyclonal (IgG) to Human TNFRSF21 / DR6
Human
ICC, Western blot
Unconjugated
Anti-TNFRSF21 / DR6 Antibody LS-C125490
Rabbit Polyclonal (IgG) to Rat TNFRSF21 / DR6
Rat, Human, Mouse
Western blot
Unconjugated
Anti-TNFRSF21 / DR6 Antibody LS-C146897
Rabbit Polyclonal to Rat TNFRSF21 / DR6
Rat, Human, Mouse
Western blot
Unconjugated

100% Guaranteed 100% Guaranteed
Rabbit Polyclonal (IgG) to Human TNFRSF21 / DR6
Human
Western blot
Unconjugated

Details

Human TNFRSF21 / DR6
Rabbit
Human (tested or 100% immunogen sequence identity)
IgG Polyclonal
Unconjugated
Affinity purified
Unmodified

Applications

Western blot (1:500)

Specificity and Use

TNFRSF21 / DR6 antibody was raised against peptide corresponding to amino acids 42-56 of human DR6 precursor.
aa42-56
Recognizes the death domain containing receptor DR6. DR6 is widely expressed in human tissues and cell lines. Overexpression of DR6 induces apoptosis and activates NF-kB and JNK.
Suitable for use in Western Blot. Western Blot: 1:500.

Packaging

PBS, 0.02% sodium azide.
Long term: -20°C; Short term: +4°C. Avoid repeat freeze-thaw cycles.
For research use only.

About TNFRSF21 / DR6

O75509 NM_014452 NP_055267.1

TNFRSF21 Antibody, CD358 antigen Antibody, BM-018 Antibody, DR6 Antibody, TNFR-related death receptor 6 Antibody, CD358 Antibody, Death receptor 6 Antibody

Promotes apoptosis, possibly via a pathway that involves the activation of NF-kappa-B. Can also promote apoptosis mediated by BAX and by the release of cytochrome c from the mitochondria into the cytoplasm. Plays a role in neuronal apoptosis, including apoptosis in response to amyloid peptides derived from APP, and is required for both normal cell body death and axonal pruning.

Requested From: United States
Date Requested: 12/3/2016

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