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Anti-TBS / SALL1 Antibody (aa29-43) LS-C112873


Wt. Vol. Conc. Price
100 µg - - Unavailable

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Goat Polyclonal to Human TBS / SALL1
Human, Mouse, Rat, Hamster


Human TBS / SALL1
Human, Mouse, Rat, Hamster (tested or 100% immunogen sequence identity)
Monkey (at least 90% immunogen sequence identity)
Immunoaffinity purified


ELISA (1:32000)

Specificity and Use

TBS / SALL1 antibody was raised against synthetic peptide EKGQPSRPTKSKDAH-C from the N-terminus of human SALL1 (NP_002959.2; NP_001121364.1). Percent identity by BLAST analysis: Human, Gorilla, Monkey, Mouse, Rat, Hamster (100%); Gibbon, Marmoset (93%); Panda, Bat, Bovine, Dog, Horse, Pig (87%); Elephant (80%).
LS-E27168 - Lyophilized - 100 µg - $145.00
Immunizing peptide used to generate LS-C112873. Useful for pre-absorption and neutralization of the antibody's antigen binding site.
Human SALL1. This antibody is expected to recognize both reported isoforms (NP_002959.2; NP_001121364.1).
Peptide ELISA: antibody detection limit dilution 1:32000. Western blot: Blot: Preliminary experiments gave an approx 100kDa band in Human Brain (Amygdala and Substantia nigra) and Mouse Kidney lysates after 1 ug/ml antibody staining. Please note that currently we cannot find an explanation in the literature for the band we observe given the calculated size of 140kDa according to Human NP_002959.2 and Mouse NP_067365.2, and 130kDa according to Human NP_001121364.1. The 100kDa band was successfully blocked by incubation with the immunizing peptide.


Tris-buffered saline, pH 7.3, 0.5% BSA, 0.02% sodium azide
Store at -20°C. Minimize freezing and thawing.
For research use only.

About TBS / SALL1

Q9NSC2 NM_002968 NP_002959.2

SALL1 Antibody, HSAL1 Antibody, Sal-like 1 (Drosophila) Antibody, SAL1 Antibody, Zinc finger protein SALL1 Antibody, ZNF794 Antibody, Sal (Drosophila)-like 1 Antibody, TBS Antibody, Zinc finger protein 794 Antibody, Zinc finger protein Spalt-1 Antibody, Sal-1 Antibody, Sal-like protein 1 Antibody

TBS / SALL1 is a zinc finger transcriptional repressor and may be part of the NuRD histone deacetylase complex (HDAC). Defects in this gene are a cause of Townes-Brocks syndrome (TBS) as well as bronchio-oto-renal syndrome (BOR). Two transcript variants encoding different isoforms have been found for this gene.

Requested From: 
Date Requested: 3/28/2017

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