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Anti-SELE / CD62E / E-selectin Antibody (Biotin) LS-C44154

Ordering

Wt. Vol. Conc. Price
- 100 tst - $395
Inquire for larger quantities

LSBio (Direct) LSBio (Direct)
206-374-1102
866-206-6909
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Most Popular SELE / CD62E / E-selectin Antibodies

Anti-SELE / CD62E / E-selectin Antibody LS-C13905
Mouse Monoclonal (IgG2a) to Human SELE / CD62E / E-selectin
Human
Immunofluorescence, Flow Cytometry
Unconjugated
Anti-SELE / CD62E / E-selectin Antibody (aa33-50) LS-C312860
Rabbit Polyclonal (IgG) to Human SELE / CD62E / E-selectin
Human, Monkey, Mouse, Rat
IHC - Paraffin, Western blot
Unconjugated
Immunohistochemistry Image

100% Guaranteed 100% Guaranteed
Mouse Monoclonal (IgG2a) to Human SELE / CD62E / E-selectin
Human
Flow Cytometry
Biotin Conjugated

Details

Human SELE / CD62E / E-selectin
Mouse
Human (tested or 100% immunogen sequence identity)
IgG2a Monoclonal
Biotin
Purified
Unmodified

Applications

Flow Cytometry

Specificity and Use

Flow Cytometry: Use 10 ul of the suggested working dilution to label 10^6 cells in 100 ul. Method sheets are available on request. The applications listed have been tested for the unconjugated form of this product. Other forms have not been tested.

Packaging

Tris-sodium chloride, 0.02% sodium azide, 1% BSA.
For research use only.

About SELE / CD62E / E-selectin

P16581 NM_000450 NP_000441.2

SELE Antibody, CD62E Antibody, ELAM Antibody, ESEL Antibody, E-selectin Antibody, LECAM2 Antibody, CD62E antigen Antibody, ELAM-1 Antibody, ELAM1 Antibody, Selectin E Antibody

SELE / CD62E / E-selectin is found in cytokine-stimulated endothelial cells and is thought to be responsible for the accumulation of blood leukocytes at sites of inflammation by mediating the adhesion of cells to the vascular lining. It exhibits structural features such as the presence of lectin- and EGF-like domains followed by short consensus repeat (SCR) domains that contain 6 conserved cysteine residues. These proteins are part of the selectin family of cell adhesion molecules.

Requested From: United States
Date Requested: 12/7/2016

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