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Anti-PRKAR2A Antibody (Pro68) LS-C176731

Ordering

Wt. Vol. Conc. Price
- 100 µl - $345
Inquire for larger quantities

LSBio (Direct) LSBio (Direct)
206-374-1102
866-206-6909
Orders@LSBio.com
 

Most Popular PRKAR2A Antibodies

Anti-PRKAR2A Antibody (clone 4H8) LS-C115181
Mouse Monoclonal [clone 4H8] (IgG1) to Human PRKAR2A
Human
IHC - Paraffin, Immunofluorescence, Western blot
Unconjugated
Immunohistochemistry Image
Anti-PRKAR2A Antibody (aa185-234) LS-C145406
Rabbit Polyclonal to Human PRKAR2A
Human, Mouse, Rat, Bovine, Dog, Horse, Pig
Western blot
Unconjugated
Western blot Image
Anti-PRKAR2A Antibody IHC-plus™ LS-B14105
Rabbit Polyclonal to Human PRKAR2A
Human
IHC - Paraffin, Western blot
Unconjugated
Immunohistochemistry - Paraffin Image

100% Guaranteed 100% Guaranteed
Rabbit Polyclonal to Human PRKAR2A
Human
IHC, Immunofluorescence, Western blot
Unconjugated

Details

Human PRKAR2A
Rabbit
Human (tested or 100% immunogen sequence identity)
Polyclonal
Unconjugated
Immunoaffinity purified
Unmodified

Applications

  • IHC
  • Immunofluorescence
  • Western blot

Specificity and Use

PRKAR2A antibody was raised against synthetic peptide around Pro68 of human PKR2 / PRKAR2A
Pro68
Human PKR2 / PRKAR2A

Packaging

PBS, pH 7.2, 0.05% sodium azide
Store at 4°C short term. Aliquot and store at -20°C long term. Avoid freeze-thaw cycles.
For research use only.

About PRKAR2A

P13861 BT007225 AAP35889.1

PRKAR2A Antibody, PKR2 Antibody, PRKAR2 Antibody

cAMP is a signaling molecule important for a variety of cellular functions. cAMP exerts its effects by activating the cAMP-dependent protein kinase, which transduces the signal through phosphorylation of different target proteins. The inactive kinase holoenzyme is a tetramer composed of two regulatory and two catalytic subunits. cAMP causes the dissociation of the inactive holoenzyme into a dimer of regulatory subunits bound to four cAMP and two free monomeric catalytic subunits.

Western blot

Western blot
Western blot of PKA II reg (P68) pAb in extracts from 293 cells.
 

Requested From: United States
Date Requested: 12/4/2016

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