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Anti-KCNQ3 Antibody (aa861-872) LS-C112723

Ordering

Wt. Vol. Conc. Price
100 µg - - Unavailable

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Goat Polyclonal to Human KCNQ3
Human, Monkey
ELISA
Unconjugated

Details

Human KCNQ3
Goat
Human, Monkey (tested or 100% immunogen sequence identity)
Polyclonal
Unconjugated
Immunoaffinity purified
Unmodified

Applications

ELISA (1:16000)

Specificity and Use

KCNQ3 antibody was raised against synthetic peptide C-SDSVWTPSNKPI from the C-terminus of human KCNQ3 (NP_004510.1). Percent identity by BLAST analysis: Human, Gorilla, Gibbon, Monkey (100%); Platypus (92%); Marmoset, Mouse, Rat, Hamster, Elephant, Panda, Bat, Horse, Rabbit, Pig, Opossum, Turkey (83%).
LS-E27010 - Lyophilized - 100 µg - $145.00
Immunizing peptide used to generate LS-C112723. Useful for pre-absorption and neutralization of the antibody's antigen binding site.
aa861-872
Human KCNQ3.
Peptide ELISA: antibody detection limit dilution 1:16000. Western blot: Preliminary experiments gave an approx. 23kDa band in Human Brain (Cerebral Cortex, Amygdala, Hippocampus) lysates after 0.3 ug/ml antibody staining. Please note that currently we can not find an explanation in the literature for the band we observe given the calculated size of 96.7kDa according to NP_004510.1. The 23kDa band was successfully blocked by incubation with the immunizing peptide.

Packaging

Tris-buffered saline, pH 7.3, 0.5% BSA, 0.02% sodium azide
Store at -20°C. Minimize freezing and thawing.
For research use only.

About KCNQ3

O43525 NM_004519 NP_004510.1

KCNQ3 Antibody, BFNC2 Antibody, KV7.3 Antibody, EBN2 Antibody, KQT-like 3 Antibody

Probably important in the regulation of neuronal excitability. Associates with KCNQ2 or KCNQ5 to form a potassium channel with essentially identical properties to the channel underlying the native M-current, a slowly activating and deactivating potassium conductance which plays a critical role in determining the subthreshold electrical excitability of neurons as well as the responsiveness to synaptic inputs.

Requested From: 
Date Requested: 3/29/2017

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