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Human KCNJ4 / Kir2.3 | |
Mouse | |
Human, Mouse, Rat (tested or 100% immunogen sequence identity) | |
IgG1 Monoclonal [S25-35] | |
Atto 655. Also available Unconjugated or conjugated with Atto 565, Atto 594, Atto 633, Atto 680, Atto 700, AP, APC, Biotin, FITC, PE, Atto 594, PerCP, RPE, SA, Atto 390, Atto 488. | |
Protein G purified from tissue culture supernatant | |
Unmodified |
KCNJ4 / Kir2.3 antibody was raised against fusion protein amino acids 390-445 of human Kir2.3, accession number NP_032453. | |
aa390-445 | |
Detects ~45kD. No cross-reactivity against Kir2.1 or Kir2.2. | |
The applications listed have been tested for the unconjugated form of this product. Other forms have not been tested. |
PBS, pH 7.4, 0.09% sodium azide, 50% glycerol. | |
Store at -20°C. | |
For research use only. |
KCNJ4 Antibody, IRK3 Antibody, Kir2.3 Antibody, HIR Antibody, HIRK2 Antibody, HRK1 Antibody, IRK-3 Antibody, Hippocampal inward rectifier Antibody
Several different potassium channels are known to be involved with electrical signaling in the nervous system. One class is activated by depolarization whereas a second class is not. The latter are referred to as inwardly rectifying K+ channels, and they have a greater tendency to allow potassium to flow into the cell rather than out of it. This asymmetry in potassium ion conductance plays a key role in the excitability of muscle cells and neurons.
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