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Anti-GPR65 / TDAG8 Antibody (aa176-225) LS-C151819


Wt. Vol. Conc. Price
50 µg - 1 mg/ml Unavailable

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Rabbit Polyclonal to Human GPR65 / TDAG8
Human, Monkey
Western blot (applications tested for the base form of this product only)


Human GPR65 / TDAG8
Human, Monkey (tested or 100% immunogen sequence identity)
Unconjugated. Also available conjugated with Biotin, FITC, HRP.
Immunoaffinity purified


  • Western blot
  • (applications tested for the base form of this product only)

Specificity and Use

GPR65 / TDAG8 antibody was raised against synthetic peptide located between aa176-225 of human GPR65 (Q8IYL9, NP_003599). Percent identity by BLAST analysis: Human, Gorilla, Gibbon, Monkey, Marmoset (100%); Chimpanzee, Galago (92%); Panda, Opossum (84%); Mouse (78%).
LS-E23620 - Lyophilized - 100 µg - $145.00
Immunizing peptide used to generate LS-C151819. Useful for pre-absorption and neutralization of the antibody's antigen binding site.
Human GPR65


Lyophilized from PBS with 2% sucrose
Distilled water
Long term: -20°C, the use of 50% glycerol is recommended if storing aliquots in -20°C for long term use (up to 1 year); Short term (less than one week): +4°C. Avoid repeat freeze-thaw cycles.
For research use only.

About GPR65 / TDAG8

Q8IYL9 NM_003608 NP_003599.2

GPR65 Antibody, G-protein coupled receptor 65 Antibody, Gpcr25 Antibody, HTDAG8 Antibody, TDAG8 Antibody, T-cell death-associated gene 8 Antibody, Psychosine receptor Antibody, G protein-coupled receptor 65 Antibody

GPR65 (TDAG8), a Lysophospholipid/Lysosphingolipid Receptor, is a molecular target for psychosine, a toxic lipid formed by the breakdown of galactosylceramide (cerebroside). A build-up of psychosine, caused by the lack of galactosylceramidase, results in the death of the myelin-synthesizing cells, oligodendrocytes, which is associated with Krabbe disease.

Western blot

Western blot
GPR65 / TDAG8 antibody Western Blot of U937. This image was taken for the unconjugated form of this product. Other forms have not been tested.

Requested From: 
Date Requested: 4/28/2017

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