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Anti-GCG / Glucagon Antibody (aa8-18) LS-C187646


Wt. Vol. Conc. Price
- 500 µl 5 mg/ml $245
Inquire for larger quantities

LSBio (Direct) LSBio (Direct)

Most Popular GCG / Glucagon Antibodies

Anti-GCG / Glucagon Antibody IHC-plus™ LS-B1919
Rabbit Polyclonal to Human GCG / Glucagon
IHC - Paraffin, Western blot
Immunohistochemistry Image
Anti-GCG / Glucagon Antibody (aa25-54) IHC-plus™ LS-B8235
Rabbit Polyclonal to Human GCG / Glucagon
Human, Mouse
IHC - Paraffin, Western blot, Flow Cytometry
Immunohistochemistry Image

100% Guaranteed 100% Guaranteed
Goat Polyclonal (IgG) to Human GCG / Glucagon


Human GCG / Glucagon
Human (tested or 100% immunogen sequence identity)
IgG Polyclonal
Protein G purified



Specificity and Use

GCG / Glucagon antibody was raised against synthetic peptide corresponding to amino acids 8-18 of human glucagon.
Detects glucagon, a pancreatic hormone whose actions are antagonistic to insulin, functioning to increase blood glucose through stimulation of glycogenolysis and gluconeogenesis in the liver. Glucagon is a ligand for the glucagon receptor, a G-protein linked receptor whose signalling pathway controls cell proliferation. Glucagon is secreted from A-cells within the Islets of Langerhans in the pancreas.


PBS, 0.09% sodium azide
+4°C or -20°C, Avoid repeated freezing and thawing.
For research use only.

About GCG / Glucagon

P01275 NM_002054 NP_002045.1

GCG Antibody, GLP2 Antibody, Glucagon Antibody, Glucagon-like peptide 2 Antibody, GRPP Antibody, Glicentin-related polypeptide Antibody, GLP1 Antibody, Glucagon-like peptide 1 Antibody

Glucagon plays a key role in glucose metabolism and homeostasis. Regulates blood glucose by increasing gluconeogenesis and decreasing glycolysis. A counterregulatory hormone of insulin, raises plasma glucose levels in response to insulin-induced hypoglycemia. Plays an important role in initiating and maintaining hyperglycemic conditions in diabetes.GLP-1 is a potent stimulator of glucose-dependent insulin release.

Requested From: United States
Date Requested: 10/25/2016

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