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Anti-BAK1 / BAK Antibody (aa20-124) LS-C125160


Wt. Vol. Conc. Price
100 µg - - $695
Inquire for larger quantities

LSBio (Direct) LSBio (Direct)

Most Popular BAK1 / BAK Antibodies

Anti-BAK1 / BAK Antibody IHC-plus™ LS-B8643
Rabbit Polyclonal (IgG) to Human BAK1 / BAK
Human, Mouse
IHC - Paraffin, Western blot
Immunohistochemistry Image
Anti-BAK1 / BAK Antibody (N-Terminus, Biotin) LS-C275410
Rabbit Polyclonal (IgG) to Human BAK1 / BAK
IHC, Western blot, ELISA
Biotin Conjugated

100% Guaranteed 100% Guaranteed
Goat Polyclonal (IgG) to Human BAK1 / BAK
Human, Mouse, Rat
Western blot


Human BAK1 / BAK
Human, Mouse, Rat (tested or 100% immunogen sequence identity)
IgG Polyclonal
Immunoaffinity purified


Western blot (1 µg/ml)

Specificity and Use

BAK1 / BAK antibody was raised against recombinant human BAK, aa20-124; Q16611 (E. coli).
Recognizes human BAK at ~29kD. Species cross Reactivity: mouse and rat.
Suitable for use in Western blot: 1.0 ug/ml.


Lyophilized from 0.2 um sterile-filtered solution in PBS, 5% trehalose
40-50% glycerol, PBS.
Lyophilized powder may be stored at 4°C for short-term only. Reconstituted product is stable for 12 months at -20°C.
For research use only.

About BAK1 / BAK

Q16611 NM_001188 NP_001179.1

BAK1 Antibody, Apoptosis regulator BAK Antibody, BAK Antibody, BCL2L7 Antibody, Bcl-2-like protein 7 Antibody, BCL2-antagonist/killer 1 Antibody, Bcl2-L-7 Antibody, BCL2-like 7 protein Antibody, CDN1 Antibody, Pro-apoptotic protein BAK Antibody, BAK-LIKE Antibody

Apoptosis plays a major role in normal organism development, tissue homeostasis, and removal of damaged cells. Disruption of this process has been implicated in a variety of diseases such as cancer. The Bcl-2 family of proteins is comprised of critical regulators of apoptosis that can be divided into two classes: those that inhibit apoptosis and those that promote cell death. Bak, a pro-apoptotic Bcl-2 family member, is an oligomeric protein that localizes to the mitochondria.

Requested From: United States
Date Requested: 10/24/2016

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