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Anti-ATP13A2 Antibody (Ser282) LS-C191165

Ordering

Wt. Vol. Conc. Price
- 100 µl - $455
Inquire for larger quantities

LSBio (Direct) LSBio (Direct)
206-374-1102
866-206-6909
Orders@LSBio.com
 

Most Popular ATP13A2 Antibodies

Anti-ATP13A2 Antibody (Internal, DY488) LS-C141384
Rabbit Polyclonal to Human ATP13A2
Human, Mouse
IHC, IHC - Paraffin, Western blot
DY488 Conjugated
Immunohistochemistry Image
Anti-ATP13A2 Antibody (aa200-300) IHC-plus™ LS-B11104
Rabbit Polyclonal to Human ATP13A2
Human, Mouse
IHC - Paraffin, Western blot
Unconjugated
Immunohistochemistry Image

100% Guaranteed 100% Guaranteed
Rabbit Polyclonal (IgG) to Human ATP13A2
Human, Monkey, Mouse, Rat
Western blot, Immunoprecipitation
Unconjugated

Details

Human ATP13A2
Rabbit
Human, Monkey, Mouse, Rat (tested or 100% immunogen sequence identity)
IgG Polyclonal
Unconjugated
Affinity purified
Unmodified

Applications

  • Western blot (1:1000)
  • Immunoprecipitation (1:50)

Specificity and Use

ATP13A2 antibody was raised against synthetic peptide corresponding to residues surrounding Ser282 of human PARK9.
Ser282
Endogenous levels of total human PARK9. Species Crossreactivity: Monkey, mouse and rat.

Packaging

10 mM sodium HEPES, pH 7.5, 150 mM sodium chloride, 100ug/ml BSA, 50% glycerol
May be stored at 4°C for short-term only. Aliquot to avoid repeated freezing and thawing. Store at -20°C. Aliquots are stable for at 12 months.
For research use only.

About ATP13A2

Q9NQ11 NM_022089 NP_071372.1

ATP13A2 Antibody, HSA9947 Antibody, PARK9 Antibody, RP1-37C10.4 Antibody, Putative ATPase Antibody, ATPase type 13A2 Antibody, CLN12 Antibody, KRPPD Antibody

ATP13A2 belongs to the P-type superfamily of ATPases that transport inorganic cations and other substrates across cell membranes. ATP13A2 has predominantly neuronal expression and transiently transfected cells studies have shown that it is located in lysosomes. Recently a loss-of-function mutation in ATP13A2 has been linked to an autosomal recessive form of early-onset parkinsonism with pyramidal degeneration and dementia.

Requested From: United States
Date Requested: 12/5/2016

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